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A 56-year-old man, originally from India and with a history of hypertension, had developed bilateral red, swollen ankles while on a recent trip to Bangalore. Five days later, the patient developed severe fever and shaking chills that resolved within 48 hrs.
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In October of 2005, the first reports of 5 cases and the possible association of Guillain-Barré Syndrome (GBS) and recipients of Menactra® Meningococcal Conjugate Vaccine (MCV4) were reported.
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Segers and colleagues of the University of Amsterdam conducted this randomized, double-blind clinical trial at a 480-bed community hospital that performs 1200 cardiac surgical procedures annually.
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In patients with M. Tuberculosis infection, the emergence of chromosomally mediated resistance to INH is a stochastic process, meaning it is the sum of the product of the spontaneous mutation rate (10-6) and the total bacterial load.
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In a multicenter international open-label study, patients with proven or probable invasive aspergillosis (IA) that was refractory to standard antifungal therapy or who were intolerant to such therapy were treated with posaconazole in a total daily amount of 800 mg administered in divided doses.
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Pronovost and colleagues conducted a multi-hospital cohort study aimed at reducing the rate of catheter-related bloodstream infections (CRBSIs) in ICUs. The intervention focused on increasing adherence to evidence-based CDC recommendations for catheter insertion.
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Male sexual partners of women with vaginal trichomoniasis were tested, using urethral and urine culture, as well as urine polymerase chain reaction (PCR), for evidence of infection with this flagellated protozoan.
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Uk-427,857 (Maraviroc) is an investigational small-molecule HIV entry inhibitor that binds to the CCR5 chemokine receptor on the surface of T-cells. This chemokine receptor is normally the receptor for the chemokines MIP-1 alpha and RANTES.
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Treatment of chronic Hepatitis B is directed at suppressing viral replication, reducing hepatitis (necroinflammatory) activity, slowing progression of fibrotic disease, inducing loss of HBeAg with seroconversion to anti-HBe, and rendering patients noninfectious.
